Archive for June, 2011

UPPER RESPIRATORY TRACT INFECTIONS: ACUTE SINUSITIS

June 25th, 2011

Sinusitis is perhaps the most misunderstood infection of the upper respiratory tract. Sinusitis is defined as inflammation of the paranasal sinuses and contiguous nasal mucosa, regardless of the cause. Acute sinusitis is among the most common conditions treated by primary care physicians and is a common reason for the administration of antibiotics. Many clinicians consider sinusitis to be primarily of bacterial origin, but this is untrue. Although acute bacterial sinusitis does occur, acute sinusitis is often viral and therefore does not necessitate treatment with antibiotics. Distinguishing viral from acute bacterial sinusitis is difficult, and this has led to the widespread overuse of antibiotics for the treatment of sinusitis.The clinical features of sinusitis include purulent nasal discharge, nasal congestion, facial pain, maxillary toothache, and occasionally fever or cough. The clinical features of viral rhinosinusitis and acute bacterial sinusitis are similar. However, acute bacterial sinusitis typically develops secondarily to viral rhinosinusitis. During a viral upper respiratory tract infection, thick nasal secretions accumulate in the sinuses. Bacterial super-infection can subsequently result. Some authors have suggested that the high intranasal pressures generated during nose blowing may be a contributing factor for the introduction of bacterial pathogens into the sinus cavities. Other causes of acute bacterial sinusitis are less common and include seasonal allergies, mechanical obstruction of sinus drainage, swimming, prolonged nasal intubation, and extension from a dental infection into the sinus cavity.No diagnostic test easily distinguishes between viral rhinosinusitis and acute bacterial sinusitis. Sinus aspiration, although performed in some research studies, is not practical for routine diagnosis of acute bacterial sinusitis. The value of radiologic studies, including computed tomographic scans of the sinuses, is also limited because radiographic studies are unable to distinguish changes due to viral rhinosinusitis from those caused by acute bacterial sinusitis.18The decision to treat patients with antibiotics most often must be made on clinical grounds alone. Acute bacterial sinusitis is unlikely in patients whose symptoms are less than 7 days in duration. Patients whose symptoms have lasted more than 7 days are more likely to have bacterial sinusitis. However, the duration of illness alone is insufficient to suggest bacterial sinusitis. As discussed earlier, up to 25% of patients with viral rhinosinusitis will still have symptoms at 14 days. Other clinical predictors that suggest acute bacterial sinusitis include maxillary tooth or facial pain (especially unilateral), unilateral sinus tenderness, and mucopurulent nasal discharge. Worsening of symptoms after initial improvement is another clue to acute bacterial sinusitis.Even if a patient is suspected of having an acute bacterial sinusitis based upon these clinical predictors, it is often not necessary to prescribe antibiotics. A recent meta-analysis showed that although there is some benefit to treating patients with acute bacterial sinusitis, the benefit is small, and resolution of symptoms typically occurs without antibiotics. Patients with mild or moderate symptoms can be treated with nasal decongestants and analgesics, as discussed in the previous section. Current recommendations suggest the use of antibiotics in patients with severe symptoms of acute bacterial sinusitis, as outlined earlier. Even if antibiotics are used, decongestants should be administered to maintain sinus drainage.The choice of initial antibiotic therapy is empiric and based on knowledge of the organisms likely to cause acute bacterial sinusitis. Failure of treatment should result in a broadening of the antibiotic spectrum and prompt consideration of diagnostic sinus aspiration.Complications of bacterial sinusitis are rare but can be serious. These complications include meningitis, brain abscess, and periorbital cellulitis. Currently, there are no data to suggest that early treatment prevents the development of these complications.The possibility of fungal sinusitis should be strongly considered in the immunocompromised host. Rhinocerebral mucormycosis is an acute invasive fungal sinusitis that typically occurs in patients with organ transplants, diabetes mellitus, or neutropenia (due to hematologic malignancy or immunosuppressive medications). It rarely affects hosts with a normal immune system. A black necrotic palatal or nasal eschar may be seen and is a clue to the diagnosis. When an invasive fungal sinusitis is suspected, therapy with intravenous amphotericin В should be initiated, and urgent surgical consultations should be obtained.*35/348/5*

POST-TRAUMATIC STRESS SYNDROME

June 14th, 2011

When stress breakdown symptoms have been experienced over a period of time in relation to one specific stress, the person may become conditioned to associate the symptoms of breakdown with memories of the stressful situation. Just as a person can be conditioned to associate unpleasant or pleasant memories with odours or sounds, the person who has undergone severe stress may re-experience some of the feelings experienced at the time of the stressful event when these are triggered by stimuli which rekindle memories of the event.Thus, a person who experienced anxiety with the sound of helicopters in a war zone may re-experience some of that anxiety at the sound of a helicopter passing overhead years later. If a person held prisoner in an airplane hijacking experienced stage three symptoms, mention of the event or associations with it might recall the memories of what the breakdown symptoms felt like.Moreover, if the person still has a lot of unresolved feelings and repressed conflicts about this stressful period of his life, then the rekindled feelings might re-introduce those emotional conflicts. This vicious cycle of after-effects can be called the post-traumatic stress syndrome.It is not the aim of this book to deal with the post-traumatic stress syndrome, a complex problem requiring those skills necessary to examine conflicts which have been repressed into the unconscious mind. The correct treatment of post-traumatic stress disorder is by skilled psychotherapy.
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EPILEPSY AND ITS SPECIAL FORMS/EPILEPSY SYNDROMES: JUVENILE MYOCLONIC EPILEPSY OF JANZ

June 3rd, 2011

Juvenile myoclonic epilepsy is a relatively newly recognized syndrome unfamiliar to many physicians who do not work in the field of epilepsy. It is easily recognized, if you know what to look for and know what questions to ask of the patient. It is also easily treated.Epilepsy of Janz starts in late childhood or adolescence, often about the time of puberty. Its hallmark is mild myoclonic jerks, most common as the person is going to sleep or awakening in the morning. An adolescent will describe jerking of the arms or legs, a feeling of being very “jumpy.” Some patients have told us that they set their alarm clocks to wake up early and then stay in bed for one half hour to an hour, until the jumpiness wears off. They say that if they get up more quickly the jerking gets much worse.If a person has early morning seizures, informing your doctor about the jerks that precede them may make it easier to diagnosis this particular form of epilepsy.Occasionally, the jerking builds up and becomes sufficiently severe so that the person experiences a clonic or a tonic-clonic seizure. In addition, people may experience absence seizures.The EEG between seizures, in this form of epilepsy, often shows a fast, multiple- or double-spike pattern followed by slow waves, with fast rapid spikes occurring during the jerks. When the diagnosis is suspected, the best way of confirming it is a sleep EEG, continued for ten or fifteen minutes after the person awakens. It is during this time that the jerks and the characteristic EEG pattern are most likely to be seen.Diagnosis is important because although this form of epilepsy responds poorly to many medications, it is usually easily controlled with valproic acid. The seizures often recur when this medication is withdrawn. A familv history of epilepsy may occur in as many as 40 percent of siblings of those with the epilepsy of Janz. Studies of these families are beginning to provide clues to its genetic basis.*92\208\8*